Joe, Joe, Joe ....
Sometimes a cigar is just a cigar, and sometimes a physical illness is just a physical illness. You need to be informed.
See this excellent link to essential tremor. My father had it. He was never on a psychotropic med in his life. He was born in 1906 for God's sake, LOL. Never believed in psychiatry. He self medicated his tremor with alcohol, NOT in excess!
Also, my tinnitus was caused by an ear infection. It's gone. I have a bad sinus which I had surgery for back in 1993. Also had my tonsils out. Infections ceased as did tinnitus. When infections return with FAR less frequency, I occasionally get tinnitus. No connection with Klonopin. A course of antibiotics eliminated it. I don't have tinnitus!!!!
Joe, you have to READ reputable sources. And I have seen not only my shrink, but my GP, a medical resident, and will be seeing a neurologist in June. The GP diagnosed "essential tremor"!
Here's stuff on essential tremor. Also, I recently have had 4 vials of blood drawn which is being analyzed for differential diagnosis which could be a thyroid problem, and about a million other things.
Benzos are used in medicine for many things. Try living with essential tremor. If you have something that works to improve quality of life, you use it. You have an informed decision to make, yes. I'm not going to increase my Klonopin, or add any Benzos myself, or start drinking, LOL. I will wait for my blood tests, and see the neurologist. Per 3 doctors so far this has NOTHING to do with any psychotropic med I'm on.... particularly Klonopin!
There are a lot of folks out there who can't function with essential tremor. And many who get it who've never been on psychiatric meds!!! Good grief. In rare cases, newborns have essential tremor!!!!
Ya' gotta believe me Joe, that this is MY way of treating myself. I'm not pushing it on anyone. I'm giving another view is all.
Causes: ET probably represents a syndrome; multiple etiologies can be identified. Most or all of these causes are probably genetic.
ET is familial in at least 50-70% of cases. Transmission is autosomal dominant, with incomplete penetrance. Some cases are sporadic with unknown etiology.
Variations in methodology (assessment procedures and diagnostic criteria) account for the wide variation in findings; reported studies have found that 17% to almost 100% of cases are familial.
One study demonstrated an increase from 67.7% to 96% of cases having an affected relative after repeated and varying questioning, followed by direct interviewing of family members. MY FATHER HAD THIS! HE HAD TO STOP BEING A SURGEON BECAUSE OF IT!!!!! I am not pleased!!!!!
Genetics: Two susceptibility loci have been found.
The FET1 gene is located at 3q13 and was identified in 75 members of 16 Icelandic families.
Another locus, 2p25-22, was identified in 15 members of 4 generations of Americans. Abnormalities found in 3 additional American families have been reported to map to this locus.
In one family with levodopa-responsive, autosomal dominant, Lewy body parkinsonism, a chromosome arm 4p haplotype that segregates with the disease was identified. This haplotype also occurred in individuals in the family who did not have parkinsonism but rather a postural tremor consistent with ET. This suggests that in some cases postural tremor can be an alternative phenotype of the same mutation.
Associations between ET and Parkinson disease (PD) and ET and dystonia have been suggested.
ET has been hypothesized to be a risk factor for the development of PD. Some patients with PD report a longstanding history of bilateral upper extremity postural tremor. Without biological markers for these diseases, determining whether longstanding postural tremor is part of a PD syndrome or reflects the presence of both ET and PD is not possible.
Some patients with focal dystonia, such as torticollis, have mild bilateral upper extremity postural tremors. Without biological markers for these diseases, determining whether postural tremor is part of a focal dystonia syndrome or reflects the presence of both dystonia and ET is not possible.
Other diagnostic considerations:
The Movement Disorders Society has proposed the following diagnostic criteria for classic ET:
Inclusion criteria are as follows:
Bilateral, largely symmetric postural or kinetic tremor involving hands and forearms that is visible and persistent
Possible additional or isolated tremor in head but absence of abnormal posturing
Exclusion criteria are as follows:
Other abnormal neurologic signs, especially dystonia
The presence of known causes of enhanced physiologic tremor, including current or recent exposure to drugs that are known to cause tremor or a drug-withdrawal state. Per my doctors, this does NOT apply to me. My genetic propensity is the suspect.
Historic or clinical evidence of psychogenic tremor
Convincing evidence of sudden onset or evidence of stepwise deterioration
Primary orthostatic tremor
Isolated voice tremor
Isolated position-specific or task-specific tremors, including occupational tremors and primary writing tremor
Isolated tongue or chin tremor
Isolated leg tremor
DIFFERENTIALS Section 4 of 9
Author Information Introduction Clinical Differentials Workup Treatment Medication Follow-up Bibliography
Caffeine - I DON'T drink this anymore because of its worsening my DP and causing the tremor to get worse.
Multiple System Atrophy
I pray to God I don't have Parkinson's. It was suggested since my mother said my father HAD Parkinson's I could have that. God help me. But my mother was nuts. She'd say things that weren't true 87% of the time.
I'll find the treatment section.
The neurological visit and bloodwork wlll be a process of elimination. My GP was about 90% certain this is essential tremor which is YES exacerbated by anxiety and stress. I'm a MESS! What can I do? Buddhist thought, yoga, some vitamin treatments -- RADICAL ACCEPTANCE. Living every day to its fullest.