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Discussion Starter · #1 ·
I'm curious to hear your thoughts.

Are they on the brink? Not even looking for it? Not big enough of a market?
Are they close with anti-epileptic drugs, i.e. klonopin/lamictal?

with in 2 years?
5-10?
never?

What do you guys think?
 

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I read somewhere the one doctor that was experimenting with lamictal said something about how they have made so much progress already he wouldnt be suprised if they had a more effective treatment in the near future. thats just opinion based i would think, i dont know if they actually have anything in the works though.
 

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My honest guess, and I dont know if everyone will find this reassuring, is that when brain-imaging psychiatric patients becomes a basis for treatment, that there will not only be a DP specific protocol, but that they will probably be able to do it with drugs we have now. I dont think this will occur for about 20 years. In between now and then, there will be the same approach - take a wild guess, give pills to people with DP/DR, and have them fill out very subjective forms on how it affected their DP. This is how Daphne Simeon does it as well as Kings College DP Unit in London. Its unscientific, unfocused, and very, very inefficient. Unfortunately, this is the way all psychiatric meds and dxes are evaluated, except for depression. They use PET studies alot for that, and the meds have gotten better because of it. For anyone who wants scanned now, really, please have a look at the Amen clinics. I'm not "plugging" them. This is the honest future of psychiatry, and this is the only way it is available now. This will be widely accepted by 2025, I think. Of course, we may stumble across something with our current methods, but really, we'll have to be mighty lucky.

http://www.amenclinic.com

Peace
Homeskooled
 

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5 or so for something that kinda works but leaves a lot to be desired, 5-10 until until we start seeing "Ask your doctor if Reali-zil is right for you" ads on TV. I'm basing that on nothing but a hunch.

Hey Homeskooled, what about Transcranial Magnetic Stimulation? Apparently frequencies less than 1 Hz inhibit brain activity, higher frequencies stimulate it. If we zap Brodmann Areas 7b, 19, and 39 with 1 Hz or less, then turn it up to 11 for areas 21 and 22, shouldn't that work?
 

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Dear Tom,
Wow, your really studying that area of the Parietal/temporal lobe. Yeah, transcranial magnetic stimulation could do the trick, but most of the studies on it are European and kind of quackish. Its also really hard to focus a magnetic field, and we dont know how it affects epilepsy for good or bad yet. But in order to KEEP the area stimulated, you would have to be wearing a helmet all day. Thus the idea of taking something orally which circulates throughout the brain all day. You might be able to buy a gadget and treat yourself with it in the morning, but I dont know what the half-life of that treatment would be...

Peace
Homeskooled
 

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Homeskooled said:
Dear Tom,
Wow, your really studying that area of the Parietal/temporal lobe. Yeah, transcranial magnetic stimulation could do the trick, but most of the studies on it are European and kind of quackish. Its also really hard to focus a magnetic field, and we dont know how it affects epilepsy for good or bad yet. But in order to KEEP the area stimulated, you would have to be wearing a helmet all day. Thus the idea of taking something orally which circulates throughout the brain all day. You might be able to buy a gadget and treat yourself with it in the morning, but I dont know what the half-life of that treatment would be...

Peace
Homeskooled
"...you would have to be wearing a helmet all day." :lol: :lol: :lol: Well, maybe if I painted it skin color and put hair on the outside, no one would notice? Or I'll just co-ordinate my wardrobe with cleats, shoulder pads, and a U of M football jersey.

"You might be able to buy a gadget and treat yourself with it in the morning..." I'm stripping an extension cord and getting out the electrical tape as we speak. Sure is tough to find your own Parietal lobe looking in a mirror though.
 

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To clarify, there is no specific link between our study and the use of any medication. Opiate antagonists are rarely given as a treatment in the general population when compared to other treatments, and a general study like this would not capture this type of data anyway.

However, the study does has a very general purpose of gathering a large amount of data in hopes to support further interest in funding any type of treatment based on subjective data of participants. I have mentioned at times that we hope that this study can provide useful data that warrants further research, however this is extremely generalized. Having a large population study is most useful as supportive evidence that a problem exists that is important and money should be spent towards researching, whether that be specific treatments or even brain mapping (or genomic mapping of the cannaboid receptor as Dr. Simeon is conducting a pilot study on).

There is current literature regarding using Naltrexone as a treatment option for Depersonalization, and I have mentioned it on a different message board. However, this is defintely not a specific "purpose" of the study.

The purpose of the study is exactly as we have described: To gather as much data as possible on drug and and non-drug induced DP/DR and hope to gain useful statistical information when we look at results. I will be able to present the data when we are complete, and everyone will see exactly what we are trying to do with the study.

Best,

David
 
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