SUBJECT>Re:kEtAnsErIn
POSTER>Cavan
EMAIL>cavan@dial.pipex.com
DATE>Wednesday, 26 January 2000, at 3:03 p.m.
Sorry if I appear to be changing my mind on a daily basis.
I think if your dpn. was induced by some serotonin 2 agonist, then obviously taking an antagonist beforehand might block it, or a little afterwards it might reverse it.
I used to think that this was enough to warrant taking the stuff for drn. and dpn. but I realise now that's probably an over-simplification.
I advocate that experimenting with agonists and antagonist of various receptors should may light on the mechanism of dpn., at least theoretically it should. The ethics of doing this in healthy people are questionable, never mind ill people, especially until more data on HPPD is around.
Its likely, as with all medications, that analytical research will play a smaller part than we are led to believe, as anti-depressants and anti-psychotics, anti-anxiety and anti-convulsants were all discovered 'by accident' in the sense that they were originally administered for other physical ailments wherby their mental affects came to light.
As a Science Communicator and 'kind of' an Artist, I try to promote the idea of creating opportunities for serendipity, since historically its always proven a quicker route to a solution than analysis, but for obvious reasons you can't really play with peoples minds and brains in this way. I don't want to 'experimently' precipitate dpn. in myself again, although I have played with drugs in the past, and have learned from doing so.
Also, I'm well at the moment, and that makes the big difference. If a couple of years ago ECT or a lobotomy might have given me the slimmest chance of recovery, I would have said yes.
ramble
Anyroad, I'm always getting my 2A/2C subtypes mixed up.
bubble-blub-blub
cAvAn